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Systemic treatment with the enteric bacterial fermentation product, propionic acid, produces both conditioned taste avoidance and conditioned place avoidance in rats

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This improvement may have been achieved by downregulating phosphorylation of cyclin-dependent kinase 5 (CDK-5) and increasing phosphorylation of protein kinase-B (PKB/AKT) and glycogen synthase kinase (GSK)-3β [18]