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trachomatis that are generated in response to low concentrations of IFN-γ are metabolically active and seem to promote continuous secretion of pro-inflammatory cytokines, a condition that might contribute to tissue scarring

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ER stress results in the activation of the unfolded protein response (UPR) pathway, which if persistent, morphs into terminal UPR, which can promote upregulation of genes involved in iron uptake, storage, and utilization, and the inhibition of genes involved in the regulation of antioxidant defense and fatty acid metabolism, sensitizing cells to ferroptosis [99–102]