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Its primary physiological functions include promoting glucose-dependent insulin secretion, enhancing hepatic glycogen synthesis, and increasing the buffering capacity of adipose tissue for lipids

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This phosphorylation enhances VE-cadherin interactions with proteins, such as β-catenin and plakoglobin, and p120 via an Src-dependent mechanism 26,146 , which weakens endothelial barriers and facilitating tumor dissemination 26,27,34,147–151