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90,91 Additionally, the activity of epigenetics-modifying enzymes and their cofactors, such as TET and α-ketoglutarate (α-KG) from the tricarboxylic acid cycle (TCA), can be influenced by abnormal metabolite levels in patients with metabolic diseases, further disrupting epigenetic regulation and exacerbating disease progression

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This mechanism may function as a “backup plan” for maintaining cysteine pools and redox homeostasis in harsh tumor microenvironments, where nutrient availability can be unstable, or during specific phases of the metastatic cascade during which high oxidative stress can occur [19]