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The key pathological hallmarks of AD include amyloid-beta (Aβ) plaque accumulation, neurofibrillary tangles of hyperphosphorylated tau, chronic neuroinflammation, oxidative stress, and mitochondrial dysfunction (Lv et al., 2020)
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For this compound the honest answer is always a mouse, and the bioavailability figure is the one least likely to be quoted at all
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