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Necroptosis can be triggered by multiple endogenous and exogenous stimuli including anticancer agents, metabolic disturbance, ischemia/reperfusion injury, and activation of death receptors such as tumor necrosis factor-α (TNF-α) receptor (TNFR), toll-like receptors (TLRs) particularly TLR3/4, as well as interferon (IFN) receptors (Wang G