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Moreover, in conditions not directly caused by flavoprotein gene mutations—such as L-2-hydroxyglutaric aciduria (L2HGA) or Friedreich’s ataxia (FRDA)—metabolic pathology often involves secondary dysfunction of FAD-dependent enzymes and redox disruption

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10.1016/j.jhep.2014.03.009 175 ZhangL.WangG.HouW.LiP.DulinA.BonkovskyH